The nerve impulse is characterised by a great influx of sodium ions through channels in the neuron's cell membrane followed by an efflux of potassium ions through other channels. The neuron is unable to fire again for a short time (known as the refractory period), which is mediated by another potassium channel. The flow through these ion channels is governed by a "gate" which is opened by either a voltage change or a chemical messenger known as a ligand (such as a neurotransmitter). These channels are another target for anticonvulsant drugs.[7]

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It's not an actual virus but the result of dehydration that occurs when switching from glucose to fat for energy. Low-carb diets generally have a diuretic effect within the first few days, meaning you lose more water and electrolytes (like potassium and sodium) in urine than normal. It happens because you're body is losing water as it turns to muscle glycogen for energy and your body’s insulin levels decrease.

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What’s the catch? The ketogenic state has been linked to increasing satiety hormones and decreasing hunger hormones — well-researched during the initial phase. But once you’re off the keto diet after 30 days, the appetite-suppressing hormones will increase significantly from your baseline. Meaning that you’re likely to feel physically hungrier than you did before you started all of this dieting nonsense.

^ Jump up to: a b c d e f g h i j k l m n o p q r s Kossoff EH, Zupec-Kania BA, Amark PE, Ballaban-Gil KR, Bergqvist AG, Blackford R, et al. Optimal clinical management of children receiving the ketogenic diet: recommendations of the International Ketogenic Diet Study Group. Epilepsia. 2009 Feb;50(2):304–17. doi:10.1111/j.1528-1167.2008.01765.x. PMID 18823325

Can I eat fried chicken on keto diet

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