Fat is calorically dense which may shrink your portion sizes if you aren’t including the right balance of healthy keto options - like low carb veggies and proteins. And smaller portion sizes with increased calories can actually end up causing you to gain weight if you’re not paying attention. So instead of using keto as a free pass to eat bacon and butter everything, pay attention to the quality of your food choices.

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You’ve probably heard about the low carb, high fat diet that’s so popular among actors and models, and with good reason: low carb diets offer proper nourishment with whole foods, while keeping your body burning fat for fuel. This is a great way to be, as it makes fat loss largely effortless! But where does this “ketogenic” word fit into the picture?
There are numerous benefits that come with being on keto: from weight loss and increased energy levels to therapeutic medical applications. Most anyone can safely benefit from eating a low-carb, high-fat diet. Below, you’ll find a short list of the benefits you can receive from a ketogenic diet. For a more comprehensive list, you can also read our in-depth article here >

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Anticonvulsants suppress epileptic seizures, but they neither cure nor prevent the development of seizure susceptibility. The development of epilepsy (epileptogenesis) is a process that is poorly understood. A few anticonvulsants (valproate, levetiracetam and benzodiazepines) have shown antiepileptogenic properties in animal models of epileptogenesis. However, no anticonvulsant has ever achieved this in a clinical trial in humans. The ketogenic diet has been found to have antiepileptogenic properties in rats.[56]
It is possible to combine the results of several small studies to produce evidence that is stronger than that available from each study alone—a statistical method known as meta-analysis. One of four such analyses, conducted in 2006, looked at 19 studies on a total of 1,084 patients.[23] It concluded that a third achieved an excellent reduction in seizure frequency and half the patients achieved a good reduction.[18]

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Physicians of ancient Greece treated diseases, including epilepsy, by altering their patients' diet. An early treatise in the Hippocratic Corpus, On the Sacred Disease, covers the disease; it dates from c. 400 BC. Its author argued against the prevailing view that epilepsy was supernatural in origin and cure, and proposed that dietary therapy had a rational and physical basis.[Note 3] In the same collection, the author of Epidemics describes the case of a man whose epilepsy is cured as quickly as it had appeared, through complete abstinence of food and drink.[Note 4] The royal physician Erasistratus declared, "One inclining to epilepsy should be made to fast without mercy and be put on short rations."[Note 5] Galen believed an "attenuating diet"[Note 6] might afford a cure in mild cases and be helpful in others.[11]
It is possible to combine the results of several small studies to produce evidence that is stronger than that available from each study alone—a statistical method known as meta-analysis. One of four such analyses, conducted in 2006, looked at 19 studies on a total of 1,084 patients.[23] It concluded that a third achieved an excellent reduction in seizure frequency and half the patients achieved a good reduction.[18]

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Without carbs, your body has to find other ways to get glucose or find some other usable form of energy that is efficient and able to supply your brain with fuel. Your storage capacity of glucose is limited, and you can become carb depleted fairly quickly. When this happens, your body starts to use dietary protein or stored protein (aka your muscles) as well as lipids to create glucose through a process called gluconeogenesis.

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The ketone bodies are possibly anticonvulsant; in animal models, acetoacetate and acetone protect against seizures. The ketogenic diet results in adaptive changes to brain energy metabolism that increase the energy reserves; ketone bodies are a more efficient fuel than glucose, and the number of mitochondria is increased. This may help the neurons to remain stable in the face of increased energy demand during a seizure, and may confer a neuroprotective effect.[56]
Advocates for the diet recommend that it be seriously considered after two medications have failed, as the chance of other drugs succeeding is only 10%.[9][31][32] The diet can be considered earlier for some epilepsy and genetic syndromes where it has shown particular usefulness. These include Dravet syndrome, infantile spasms, myoclonic-astatic epilepsy, and tuberous sclerosis complex.[9][33]
Does the ketogenic diet protect the brain? Animal studies and some uncontrolled studies in humans have looked at the effect of the keto diet on a variety of conditions and show some hope that the keto diet may have some role in the treatment of other neurologic disorders (39).  Alzheimer’s Disease, Parkinson’s Disease, traumatic brain injury and stroke have all been suggested to benefit from the keto diet. And other animal studies suggest that there may also be a role of the keto diet in the treatment of some cancers (40,41,42,43).

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Using fat for fuel is often used interchangeably with using ketones, but this is not exactly the same thing. Even when you are eating a lot of carbs in your diet, you can still use fat (and do quite frequently!) as a source of energy without needing to go into ketosis. The type of fat you are using for this is primarily coming from stored fat (fatty acids). Stored fat is actually a preferred source of energy in-between meals, during rest, and after long duration exercise.  Your body has an almost unlimited capacity to store and use fat compared to carbs, as we’ve seen with the obesity epidemic. And using fat gives you twice as much energy for the same amount of work (one gram of fat is 9 calories whether you eat it or burn it, compared to carbs that provide only 4 calorie a gram) – making it a desirable source of fuel.
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There are many ways in which epilepsy occurs. Examples of pathological physiology include: unusual excitatory connections within the neuronal network of the brain; abnormal neuron structure leading to altered current flow; decreased inhibitory neurotransmitter synthesis; ineffective receptors for inhibitory neurotransmitters; insufficient breakdown of excitatory neurotransmitters leading to excess; immature synapse development; and impaired function of ionic channels.[7]

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